https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:34396 Wed 09 Mar 2022 16:03:27 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:25077 Sat 24 Mar 2018 07:15:02 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29834 Fri 22 Apr 2022 10:24:15 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:46137 n = 149). Assessments occurred at 20 months post-stroke, using the Mini Mental State Examination and serum blood levels of cholesterol, low density lipoprotein, high density lipoprotein and serum triglycerides. Results: Hierarchical linear regression showed that only acute functional impairment significantly contributed to long-term cognitive impairment in stroke survivors. Only 12% of the sample showed healthy cholesterol levels while the remaining patients showed borderline or high cholesterol levels. In terms of BMI, only 2% of the sample were underweight, 38% were within healthy range and 26% were overweight/obese. Only eight women and four men were smokers, therefore our sample of smokers was likely too small to detect any differences between smokers and non-smokers in regard to cognitive outcomes. Conclusion: Serum cholesterol, low density lipoprotein, high density lipoprotein, serum triglycerides, BMI or smoking status did not influence cognitive outcomes in older stroke surviving individuals. These findings suggest that modification of these factors may not influence cognitive outcomes in stroke-surviving individuals however should be interpreted as preliminary given limitations in the current study.]]> Fri 11 Nov 2022 16:31:53 AEDT ]]>